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NiONPs-induced alteration in calcium signaling and mitochondrial function in pulmonary artery endothelial cells involves oxidative stress and TRPV4 channels disruption

ABD

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Industry: Nanomedicine
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Instrument: Vasco, Wallis

Industry: Nanomedicine

DOI: https://doi.org/10.1080/17435390.2022.2030821

Keywords: Nickel oxide nanoparticles (NiONPs), pulmonary endothelial cells, reactive oxygen species, calcium signaling, mitochondrial dysfunction

In New Caledonia, anthropic activities, such as mining, increase the natural erosion of soils in nickel mines, which in turn, releases nickel oxide nanoparticles (NiONPs) into the atmosphere. Pulmonary vascular endothelial cells represent one of the primary targets for inhaled nanoparticles. (252 characters)
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